Abstract:Ambient fine particulate matter (PM2.5) in Guangzhou and Shenzhen were collected and analyzed during days in April, July, October 2004 and January 2005, and 17 PAHs concentrations and the toxicity equivalence concentrations (BaPeq) using benzo [a] pyrene (BaP) as the primary representative of their toxicity were characterized; inhalation cancer risk levels were calculated through linear dose-response model, and then, the sources of cancer risks were studied using Pearl River Delta (PRD) source profile and CMB receptor model. PAHs concentration were 5.87~63.36ng/m3, with the average 28.15ng/m3 in Guangzhou, and 32.68ng/m3 in Shenzhen ambient air, while it was higher in winter and lower in summer; 2.78% and 5.56% samples exceeded GB3095-1996 daily average standard, respectively; and 50% and 61.1% samples exceeded WHO daily average standard. Average inhalation cancer risk level in PRD area was in the range of 1×10-6~1×10-5, which was higher than risks from everyday life, but lower than minimum level for notification. Besides, 3 sources of inhalation cancer risk were apportioned from CMB model, which were coal burning, vehicle emission and biomass burning, among which coal burning made the largest contribution, and biomass burning was much higher in autumn and winter than in spring and summer. The contributions to both OC and BaPeq were very similar.
胡珊, 张远航, 魏永杰. 珠江三角洲大气细颗粒物的致癌风险及源解析[J]. 中国环境科学, 2009, 29(11): 1202-1208.
HU Shan, ZHANG Yuan-Hang, WEI Yong-Jie. Cancer risk level and source apportionment of ambient fine particulate matter in Pearl River Delta.. CHINA ENVIRONMENTAL SCIENCECE, 2009, 29(11): 1202-1208.